原文在这儿

http://www.nature.com/ng/journal/vaop/ncurrent/full/ng.2456.html

This is a good work.  Key word is variants, such as SNPs. 

Very large samples: 6,609 never-smoking Asian female lung cancer cases and 7,457 controls 

new susceptibility loci found:

10q25.2

6q22.2 and 6p21.32 (ROS1 gene)

previously identified loci confirmed:

5p15.33

3q28

17q24.3

A point to make here - the two most known gene alterations that cause as much as 35% of lung adenocarcinoma in Asian women, EGFR and ALK, are located on chromosome 7 and 2 respectively.  It is reasonable to draw side conclusion from such large study, that EGFR mutation or amplification, as well as xxxx-ALK fusion, as root causes of nonsmoker lung cancer,  are not inherited susceptibility.  On the other hand, the cause of CD74-ROS1 driven lung cancer might have significant family genetic contribution.  A friend who's cancer is ROS1 positive just suggested that his family be tested for susceptibility, after learning of this recent study.

RET oncogene is on 10q11.2, K-ras on 12, MET and BRAF on 7, so it is not likely the other 4 loci situated a known lung cancer oncogene.